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Autoradiograms showed that at P12 and P35 the weaver tissue contains six- to tenfold more D1.1 than normal tissue. Paper-5866313.
The number of VEGF-immunoreactive neurons in DRG but not in SCG, decreased postnatally and reached adult levels (34%) at P12. Paper-8675027.
Relationship of strain-dependent susceptibility to experimentally induced acute pancreatitis with regulation of Prss1 and Spink3 expression. Paper-15074723.
These results suggest that regulation of Prss1 and Spink3 expression is involved in the susceptibility to experimentally induced pancreatitis. Paper-15074723.
All these results suggest that before the death of the Purkinje cell by P12, there is an impaired maturation of these neurons provoked by the Lurcher gene action. Paper-7444935.
At P12, P47 and in the adult, analyses revealed a significant increase in the synapse-to-neuron ratio in synRas mice which correlated with the strength of transgene expression. Paper-11106567.
One eye was injected intravitreally with 150 ng in 1.5 microL of sEphB4 or sEphrinB2 on P12 and P14, while contralateral eyes were injected with IgG antibody as control. Paper-13715451.
In the 3 kb Spink3 promoter region, 92 or 8 nucleotide changes were found in JF1 or C3H vs C57BL/6J, respectively, whereas in the Prss1 promoter region 39 or 46 nucleotide changes were found in JF1 or C3H vs C57BL/6J, respectively. Paper-15074723.
In Western blot analyses of transgenic retinal homogenates, expression of the endogenous rhodopsin gene was detected from post-natal day (P)8; however, EGFP expression in transgenic retinas was initially detected at P12, indicating delayed expression of the transgene. Paper-11221118.
Furthermore, expression of epidermal growth factor receptor ( EGFR) and its ligands; serine protease inhibitor Kazal type 3 ( Spink3) and transforming growth factor alpha ( TGF alpha) and activation of K-Ras (GTP-Ras formation), which are frequently observed in human PDA, were indeed observed in parallel with TCs formation. Paper-15319187.

These synonyms are used for gene Spink3 (serine peptidase inhibitor, Kazal type 3): Serine protease inhibitor Kazal-type 3, Prostatic secretory glycoprotein, prostatic secretory glycoprotein, P12, p12, MGC107555.

These accession numbers are used for gene Spink3: Q5M9M3 (UNIPROT__AC), CA481615 (NCBI_GENBANK__AC), AK007985 (NCBI_GENBANK__AC).

Spink3 is a homologue of Spink3 (serine peptidase inhibitor, Kazal type 3) from Rattus norvegicus.
Spink3 is a homologue of SPINK1 (serine peptidase inhibitor, Kazal type 1) from Homo sapiens.
Spink3 is a homologue of SPINK1 (serine peptidase inhibitor, Kazal type 1) from Pan troglodytes.
Spink3 is a homologue of SPINK1 (serine peptidase inhibitor, Kazal type 1) from Canis lupus familiaris.
Spink3 is a homologue of SPINK1 (serine peptidase inhibitor, Kazal type 1) from Bos taurus.
Spink3 is a homologue of Spink1 (serine peptidase inhibitor, Kazal type 1) from Rattus norvegicus.

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